By M. Kamrava
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Finally, continued efforts in vaccine development to control C. trachomatis genital infections would seem prudent to prevent sequelae of unresolved or untreated infections of the female genital tract that can have profound effects on fertility in women. 9. References Aghaizu A, Atherton H, Mallinson H, Simms I, Kerry S, Oakeshott P, Hay PE. 2008. Incidence of pelvic inflammatory disease in untreated women infected with Chlamydia trachomatis. , Dutta, R. ,Bhengraj, A. , Salha, S. , (2009) Protective or pathogenic immune response to genital chlamydial infection in women--a possible role of cytokine secretion profile of cervical mucosal cells.
2009. Quantitative variations in the vaginal bacterial population associated with asymptomatic infections: a real-time polymerase chain reaction study. Eur J Clin Microbiol Infect Dis 28:281-5 Bjartling C, Osser S, Persson K. 2007. Deoxyribonucleic acid of Chlamydia trachomatis in fresh tissue from the Fallopian tubes of patients with ectopic pregnancy. Eur J Obstet Gynecol Reprod Biol 134:95-100 Blandau RJ, Boling JL, Halbert S, Verdugo P. 1975. Methods for studying oviductal physiology. Gynecol Invest 6:123-45 Blazar AS, Hogan JW, Seifer DB, Frishman GN, Wheeler CA, Haning RV.
2010; Risser & Risser 2007; Simms & Horner 2008). In a comprehensive study that evaluated all available original research and assessed the incidence of PID following C. trachomatis infection, it was concluded that no study could adequately answer the question and that many studies either had inaccuracies, validition problems or only indirect evidence to support their reported incidences (Risser and Risser, 2007). A similar review of the literature was undertaken by Simms and Horner (2008) who stated that a reasonable estimate of PID incidence in untreated women after C.